You're not wrong about factory food - I also agree with Lustig on metabolic disfunction being the real epidemic, and obesity merely being a symptom.

That said, it's not clear that metabolic disfunction is anything more than a necessary (but not sufficient) condition for Alzheimer's. We don't have an agreed model for the disease - the only agreement seems to be that it's the end result of a long process. As I understand it, there are two models:

- T3DM, probably starting with inactivation of GLUT1 receptors (glucose transporters triggered by insulin) in the astrocytes (glial cells that convert glucose into glycogen that the neurons then use)

- Inflammation, coming as a reaction (or over reaction) to some kind of assault., which could be virus/bacteria/mycotoxin/environmental pollution.

Thinking about it, inflammation may turn out to be simply a kind of TBI (or you could say both are BI).

There's also a strong probability that misfolded proteins have a part to play. They might arise from neurons dying from starvation (T3DM) or some kind of brain injury (TBI or inflammation). The issue is that they can spread the damage from region to region (like CJD prions).

So, snacking. Cells deal with misfolded proteins by consuming them (autophagy). This never happens if there's a constant supply of glucose - constant snacking is a good way to ensure a constant supply of glucose.